It's Not By Eating Too Much :reasons of weight rebound

Brief

Popular methods of losing weight are in essence based on two principles: burning fat or reducing calorie intake.

Doing exercises may consume a lot of energy. When vital energy is insufficient to meet the demand in sports, energy stored in fat will be used. Weight will be reduced at this time but the premise is continual exercise in a long term. Sliming pills containing lipase inhibitor, orlistat, prevent prandial fat being absorbed so as to lose weight yet rebound still takes place.

Going on diet or weight-reducing menu help people lose weight by reducing the intake of calorie but eating is turned into be monotonously mechanical, which is a pain for many people. Pills containing appetite inhibitor like sibutramine that have effects on nervous centralis in brain to stimulate a sense of satiety. However, experiments revealed that the weight of two thirds of the subjects rebound.

Why Weight Rebound Unavoidable Is When Taking Popular Methods

Popular methods of losing weight, no matter what it is, are in essence based on nothing but one of two principles: burning fat or reducing calorie intake.

1. Burning fat

Various exercises and some slimming pills are based on the principle of burning fat. The direct objective of these methods is evidently to consume energy stored in fat but not to ameliorate transformability. Exercise has some effects on bettering transformability, yet its effects are unsatisfactory. Rather than improving transformability, slimming products (particularly instant slimming pills) sold in market, some with such a quick effect, worsen organic functions and transformability. This explains why weight rebounds to a higher level when people stop taking slimming pills.

(1) Exercises

It is known that people consume energy when doing exercise. In the event that vital energy stored in human body is not sufficient to fulfill the energy demand in sports, it will turn to use energy stored in fat so weight will be reduced. However, the premise for success is continual exercise in a long run.

Exercise is conducive to improve transformability. When doing exercise, people will experience faster heartbeat and blood circulation which do good to ameliorate organic functions and consequently boost transformability. However, it has a limited effect. Furthermore, different exercises cannot achieve the same effect on transformability as transformability depends on organic functions and their internal balance. Exercise has direct influences on heart and lung yet other organs need the intermediation of blood to receive its full benefits. Indirect as its impact is, only continual exercise is effective. Once-ameliorated transformability may be quickly offset by bad habits during exercise. This accounts for why some people remain plump though they do sports every day and why most people gradually resume weight after stopping exercise. This phenomenon was considered as insufficient consumption in previous logic. In fact, the real cause lies in weak transformability.

(2) Orlistat

Slimming pills with orlistat as ingredient accounts for 42% of global market. Orlistat is a lipase inhibitor, preventing prandial fat being absorbed by human body. As it has no effect on brain and there is no need to control appetite, this kind of products are popular among overweight people. What merits attention is that it reduces the absorption of energy instead of the energy intake. Experiments reveal that orlistat may reduce about 30% the absorption of fat. This part has been released directly. Food with a high fat content needs avoiding when talking orlistat otherwise ill results such as diarrhea may come along. Besides, orlistat unavoidably may inhibit human body from absorbing necessary fatty acids (for instance EPA, DHA) and fat soluble vitamins (A, D, E etc.) when it prevents absorption of fat. Some reports say consequences such as dry and bagged skin or more seriously, cancer, heart disease or mental decline may be possible. Apparently, these reports exaggerate one aspect of orlistat’s many possibilities. Fairly speaking, orlistat is quite safe compared with other slimming pills. What people care about most is whether weight will rebound when they stop orlistat. Experts of Roche Pharmaceuticals Ltd answer the question: “It is difficult to reduce weight effectively; it is even more difficult to keep weight from rebounding. Clinical researches indicate that taking orlistat for two years may stop weight rebound.” Why two years? No convincing answer was provided. From its working principle, orlistat’s function of preventing fat absorption will fade when patients stop. Theatrically, rebound is inevitable for orlistat does not directly ameliorate human body’s transformability .

There are also slimming pills with diuretic and cathartic as ingredients. Instead of burning fat, these medicines accelerate the catabolism of water and protein, giving a misguided impression of weight loss. They cater for people who are eager to reduce weight in a short time. As a matter of fact, these medicines quicken the decrease of transformability. Therefore, weight rebound is 100% and takers will be weightier than before.

2. Reducing calorie intake

Generally, reducing intake of calorie by inhibiting appetite with the help of medicine or forced dieting is workable for losing weight. A decrease in total amount of energy intake results in a decline in fat transformation. At this time, people look slim.

(1) On diet or weight-reducing menu

As is known to all, going on diet is not the best way to lose weight. As energy intake is insufficient, organic functions and transformability will decline accordingly thus people will be weaker and weaker. When normal diet comes back, weight will rebound naturally.

Weight-reducing menu loses weight by matching nutrition appropriately and lessoning food containing high calorie content. A combination of nutrient menu and exercise is acknowledged as the ideal and healthiest means to lose weight. However, this does not mean that weight-reducing menu may improve transformability. What’s more, a glaring weakness of this method is that eating is altered to be monotonously mechanical. For most people, the sacrifice of enjoying delicious food is equal to deprivation part of pleasures in life.

In addition, with varying levels, many so-called nutritionists are made for market demand. It is advisable to consider all aspects of the trophic therapy carefully. Unscientific menu is likely to do harm to health.

(2) Sibutramine

Medicines inhibiting appetite with sibutramine as ingredient occupy a market share of 26.8% in the US, second to orlistat. Sibutramine is a typical appetite inhibitor with main effects on nervous centralis in brain. It inhibits the absorption of noradrenalin and serotonin in order to stimulate the sense of satiety. In this way, appetite can be controlled. Seen from the results of several large-scale experiments conducted in American and European countries, sibutramine bears obvious fruits. For example, in a two-year long experiment with a population of 605 adiposity patients, subjects are treated by a comprehensive therapy combining sibutramine hydrochloride (10mg/d) and low-fat diet. Six months later, 467 patients (77%) began to lose weight at a rate of=5%. 18 months after ceasing treatment, about 43% patients who had lost weight kept their weight from rebound. In other word, about 43% patients succeeded. Why more than a half of patients failed to keep their weight? Apparently sibutramine hydrochloride just reduces the total amount of energy intake but not ameliorates transformability. Why did 33% patients succeed? (It was so at least in two-year’s duration.) This success does not mean sibutramine has a direct effect on improving transformability because the experiment used a comprehensive treatment including low-fat diet as instrument so its result was influenced by several factors. Judging from medical mechanism of sibutramine, the sense of satiety will disappear and appetite will return to normal level when people stop taking sibutramine. At the same time, transformability hasn’t been essentially improved, thus, weight rebound is doomed to happen. In spite of people’s lingering fear about its side effects (such as headache, dry mouth, constipation, insomnia and growing blood pressure, etc.) and competitors’ flourishing negative comments, its probability of doing harm is actually quite low. Since the first clinical research on sibutramine, there has reported 43 deaths of sibutramine users so far, with 28 cases in America, 2 in Italy, 2 in Britain, 1 in South Africa and 1 in Switzerland. The reported death rate is 2 cases in 100,000 users per year, which is lower than the 1 % natural death rate of adiposity patients.

We don’t oppose (though we don’t advocate) taking slimming pills like orlistat or sibutramine because they are needed by serious adiposity patients. What we emphasize here is that we cannot just depend on inhibiting appetite or cutting down absorption of fat to lose weight. After all, it is a superficial therapy with no theoretic evidence to prove that the weight will not rebound. The essence lies in an enhancement of transformability.

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